Alexander Flügel

Dear visitor to our homepage,

At the Institute for Neuroimmunology and Multiple Sclerosis Research we investigate the causes and effects of autoimmune diseases, in particular multiple sclerosis. The central aim of our work is to understand the invasion of pathogenic immune cells into the central nervous system and the consequential damage to the nervous tissue.

Our ultimate goal is that our work contributes to the development of new therapeutic approaches to treat multiple sclerosis.

Below and by clicking on the headings at the top of this page you can find out about our team, our research projects and our teaching activities. We hope you enjoy our homepage: may it deepen your interest in the field of neuroimmunology!

Please do not hesitate to contact us if you have any questions.

Best regards
Prof. Dr. Alexander Flügel
Director

Institute news

event

Scientific talk: Prof. Matthias Gunzer

16:00 h. Title: "The role of neutrophils in post stroke immune suppression". Host: Alexander Flügel

At von-Siebold-Str. 3A, Göttingen or online

event

Scientific talk: Dr. Naoto Kawakami

16:00 h. "A genome-wide CRISPR screen to identify key regulators of encephalitogenic T cell infiltration into the CNS". Host: A. Flügel. Hybrid.

news

2nd funding period granted for CRC Transregio 274

The Collaborative Research Centre Transregio 274: “Checkpoints of Central Nervous System Recovery” will be funded by the DFG for a further four years.

event

Online scientific talk: Dr. Simone Brioschi, USA

16:00 h. Title: "So close and yet so different; mechanisms orchestrating development and specification of CNS macrophages". Host: Francesca Odoardi

Gerhard Pfefferle (left), Chairman of the Klaus Faber Stiftung, with Dr. Leon Hosang at the kick-off event for the Klaus Faber Fellow in 2021
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Klaus Faber Fellow cont.

Prolongation given to Dr. Leon Hosang’s reasearch fellowship from the Klaus Faber Stiftung

publication

New publication: Hosang et al. BBA - Mol Cell Res 2023

2-Methoxyestradiol-3,17-O,O-bis-sulfamate inhibits store-operated Ca2+ entry in T lymphocytes and prevents experimental autoimmune encephalomyelitis.

Images from the Institute